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<article article-type="research-article" dtd-version="1.3" xmlns:mml="http://www.w3.org/1998/Math/MathML" xmlns:xlink="http://www.w3.org/1999/xlink" xmlns:xsi="http://www.w3.org/2001/XMLSchema-instance" xml:lang="ru"><front><journal-meta><journal-id journal-id-type="publisher-id">pmj</journal-id><journal-title-group><journal-title xml:lang="ru">Тихоокеанский медицинский журнал</journal-title><trans-title-group xml:lang="en"><trans-title>Pacific Medical Journal</trans-title></trans-title-group></journal-title-group><issn pub-type="ppub">1609-1175</issn><publisher><publisher-name>TGMU</publisher-name></publisher></journal-meta><article-meta><article-id custom-type="elpub" pub-id-type="custom">pmj-793</article-id><article-categories><subj-group subj-group-type="heading"><subject>Research Article</subject></subj-group><subj-group subj-group-type="section-heading" xml:lang="ru"><subject>НАБЛЮДЕНИЯ ИЗ ПРАКТИКИ</subject></subj-group><subj-group subj-group-type="section-heading" xml:lang="en"><subject>PRACTICE OBSERVATIONS</subject></subj-group></article-categories><title-group><article-title>УЧАСТИЕ ГЛУБИННЫХ СТРУКТУР ГОЛОВНОГО МОЗГА В РАЗВИТИИ ЭПИЛЕПТИЧЕСКОГО РАЗРЯДА</article-title><trans-title-group xml:lang="en"><trans-title>ROLE OF DEEP BRAIN STRUCTURES IN OCCURRING EPILEPTIC DISCHARGE</trans-title></trans-title-group></title-group><contrib-group><contrib contrib-type="author" corresp="yes"><name-alternatives><name name-style="eastern" xml:lang="ru"><surname>Гуляев</surname><given-names>С. А.</given-names></name><name name-style="western" xml:lang="en"><surname>Gulyaev</surname><given-names>S. A.</given-names></name></name-alternatives><email xlink:type="simple">sergruss@yandex.ru</email><xref ref-type="aff" rid="aff-1"/></contrib></contrib-group><aff-alternatives id="aff-1"><aff xml:lang="ru"><institution>Тихоокеанский государственный медицинский университет</institution><country>Россия</country></aff><aff xml:lang="en"><institution>Pacific State Medical University</institution><country>Russian Federation</country></aff></aff-alternatives><pub-date pub-type="collection"><year>2013</year></pub-date><pub-date pub-type="epub"><day>28</day><month>09</month><year>2013</year></pub-date><volume>0</volume><issue>3</issue><fpage>91</fpage><lpage>94</lpage><permissions><copyright-statement>Copyright &amp;#x00A9; Гуляев С.А., 2013</copyright-statement><copyright-year>2013</copyright-year><copyright-holder xml:lang="ru">Гуляев С.А.</copyright-holder><copyright-holder xml:lang="en">Gulyaev S.A.</copyright-holder><license xml:lang="ru" license-type="creative-commons-attribution" xlink:href="https://creativecommons.org/licenses/by/4.0/" xlink:type="simple"><license-p>Данная работа распространяется под лицензией Creative Commons Attribution 4.0.</license-p></license><license xml:lang="en" license-type="creative-commons-attribution" xlink:href="https://creativecommons.org/licenses/by/4.0/" xlink:type="simple"><license-p>This work is licensed under a Creative Commons Attribution 4.0 License.</license-p></license></permissions><self-uri xlink:href="https://www.tmj-vgmu.ru/jour/article/view/793">https://www.tmj-vgmu.ru/jour/article/view/793</self-uri><abstract><p>Представлен анализ показателей видеоэлектроэнцефалог-рафического мониторинга сна больной с идиопатическими генерализованными тонико-клоническими припадками сна. На электроэнцефалограмме во второй фазе глубокого сна выявлена независимость возникновения генерализованной эпилептиформной активности от «сонных веретен» и установлено сохранение структуры «сонных веретен» даже в момент регистрации эпилептиформного паттерна. Указанные факты противоречат основному постулату теории таламокортикаль-ного взаимодействия - трансформации «сонного веретена» в комплексы «спайк - волна» и «острая - медленная волна». Наблюдение из практики наглядно демонстрирует тот факт, что возникновение на электроэнцефалограмме ритмов высокоамплитудных комплексов «спайк - волна» и «острая - медленная волна» у больных с идиопатической генерализованной эпилепсией является исключительно корковым феноменом. Возможно, он провоцируется активностью «сонного веретена», но распространение возбуждения при этом идет исключительно по корковым структурам.</p></abstract><trans-abstract xml:lang="en"><p>The paper provides analysis of values of videoelectroencephalographic monitoring during sleep of patient with idiopathic generalised tonic-clonic seizures. The electroencephalogram of the second stage of deep sleep demonstrates generalised epileptiform activity that occurs independently of the ‘sleep spindles’ and detects that the structure of ‘sleep spindles’ is kept even when the epileptiform pattern is being recorded. These facts prove to be inconsistent with the key thalamocortical relationship theory postulate, which is a transformation of ‘sleep spindles’ into ‘spike-wave’ and ‘sharp-slow wave’ complexes. The case follow-up reliably confirms that the rhythms of high amplitude ‘spike-wave’ and ‘sharp-slow wave’ complexes known to occur in the electroencephalograms of patients with idiopathic generalised epilepsy are only a cortical phenomenon likely to be caused by ‘sleep spindles’ activity. The incitation spreads by cortical structures, only.</p></trans-abstract><kwd-group xml:lang="ru"><kwd>электроэнцефалография</kwd><kwd>эпилептиформная активность</kwd></kwd-group><kwd-group xml:lang="en"><kwd>«сонные веретена»</kwd><kwd>electroencephalography</kwd><kwd>'sleep spindles’</kwd><kwd>epileptiform activity</kwd></kwd-group></article-meta></front><back><ref-list><title>References</title><ref id="cit1"><label>1</label><citation-alternatives><mixed-citation xml:lang="ru">Полыковский М.Г., Добронравов С.Н. Электроэнцефалография при кожевниковской эпилепсии // Ж. невропатологии и психиатрии. 1952. Т. 2, вып. 5. 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